By Anish Bhardwaj, Jeffrey R. Kirsch
The scientific administration of sufferers with acute mind and spinal twine damage has advanced considerably with the appearance of latest diagnostic and healing modalities. Editors Bhardwaj and Kirsch provide you with administration of Acute mind and Spinal twine damage, a brand new stand-alone connection with aid modern day neurologists and neurosurgeons maintain abreast of all of the contemporary developments in mind and spinal twine damage. Divided into 5 sections, mind harm, ischemic stroke, intracerebral and subarachnoid hemorrhage, annoying damage and clinical administration of spinal wire accidents, this article provide you with a precis of the most up-tp-date clinical technological know-how for the medical administration of sufferers with acute mind and spinal twine accidents.
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Additional resources for Acute Brain and Spinal Cord Injury: Evolving Paradigms and Management (Neurological Disease and Therapy)
Cranioplasty: cosmetic or therapeutic? Surg Neurol 1997; 47(3):238–241. 35. Schiffer J, Gur R, Nisim U, et al. Symptomatic patients after craniectomy. Surg Neurol 1997; 47(3):231–237. 36. Polin RS, Shaffrey ME, Bogaev CA, et al. Decompressive bifrontal craniectomy in the treatment of severe refractory posttraumatic cerebral edema. Neurosurgery 1997; 41(1):84–92 (discussion 92–84). 37. Yang XJ, Hong GL, Su SB, et al. Complications induced by decompressive craniectomies after traumatic brain injury.
This technique relies on ultrasonic detection of intravenously injected, gas-filled, encapsulated, microbubble contrast agents that possess microvascular rheologic characteristics similar to those of RBCs. To assess perfusion, microbubbles are infused intravenously and reach a steady state within the microcirculation. The microbubbles are then destroyed by a high-power ultrasonographic pulse, and the subsequent rate and extent of microbubble replenishment are used to determine microvascular blood velocity and volume, respectively (Fig.
Types of Structural Primary Brain Injury Focal Hematoma Epidural Subdural Intracerebral Contusion Concussion Lacerations Diffuse Concussion Multifocal contusion Diffuse axonal injury brain injury, referring to the clinical sequelae that result directly from the initial injury, can be broadly divided into focal and diffuse injuries (Table 2). Focal injuries include traumatic intracranial hematomas and contusions. Diffuse injuries comprise the clinical spectrum from concussion to posttraumatic coma or diffuse axonal injury.