By Antonio L. Bartorelli, Giancarlo Marenzi, Martin B. Leon
Contrast nephropathy is a huge and transforming into challenge. it's the 3rd most typical explanation for in-hospital acute renal failure and is linked to major in-hospital mortality, long term mortality, elevated possibility of in-hospital significant antagonistic cardiac occasions in addition to lengthy sanatorium remain and elevated expenditures of future health care.
The first of its style to debate the possibly mortal issues of distinction agent-induced nephropathy, this significant paintings specializes in the problem of distinction nephropathy in sufferers present process diagnostic and interventional strategies within the cardiac catheterization laboratory, reminiscent of coronary and peripheral angiography, percutaneous coronary and peripheral interventions.
Bringing jointly specialists from the specialties and subspecialties of cardiology, interventional cardiology, radiology and nephrology, Contrast-Induced Nephropathy is an invaluable source not just for interventional cardiologists but in addition for different execs thinking about the care of high-risk sufferers present process contrast-associated systems.
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Extra info for Contrast-Induced Nephropathy in Interventional Cardiovascular Medicine
43. Cochran ST, Wong WS, Roe DJ. Predicting angiography-induced acute renal function impairment: clinical risk model. AJR 1983; 141:1027–33. 44. Dharnidharka VR, Kwon C, Stevens G. Serum cystatin C is superior to serum creatinine as a marker of kidney function: a meta-analysis. Am J Kidney Dis 2002; 40:221–6. 45. Time course of serial cystatin C levels in comparison with serum creatinine after application of radiocontrast media. Clin Nephrol 2004; 61:98–102. 46. Page MK, Bukki J, Luppa P et al.
CIN is no different from acute renal failure of any other etiology in terms of the symptoms, signs, complications that may ensue, and prognostic implications. It is the third most common cause of acute tubular necrosis in patients admitted to hospital. 6–8 Proteinuria and oliguria may be observed in some patients, especially in those with prior renal insufficiency. However, CIN is usually non-oliguric and reversible. 7 In high-risk patients, CIN may present as a more severe acute renal failure.
J Am Coll Cardiol 2004; 44:1393–9. 89. Huber W, Schipek C, Ilgmann K et al. Effectiveness of theophylline prophylaxis of renal impairment after coronary angiography in patients with chronic renal insufficiency. Am J Cardiol 2003; 91:1157–62. 90. Manske CL, Sprafka JM, Strony JT et al. Contrast nephropathy in azotemic diabetic patients undergoing coronary angiography. Am J Med 1990; 89:615–20. 91. Maeder M, Klein M, Rickli H et al. Contrast nephropathy: review focusing on prevention. J Am Coll Cardiol 2004; 44:1763–71.